含氫電解水治療四氯化碳誘導的肝臟損傷

 

這是來自台灣中山醫科大學的研究,研究發現飲用電解水可以保護小鼠四氯化碳誘導的肝臟損傷。研究採用慢性損傷模型,每天給動物飲用8小時電解水。觀察了肝功能有關的轉氨酶,幾種與抗氧化酶和組織形態學改變。研究結果表明,含氫電解水可減輕四氯化碳誘導的肝臟損傷。研究還有一個肝保護的陽性對照組 水飛薊組Silymarin 200 mg/kg)。

過去電解水方面的研究都提出“活性氫”的概念,這個文章用氫氣來解釋,是一個進步。因此把這個文章也在這裡討論。

從研究方法上看,這個文章比較一般,台灣這個學者過去一直研究天然抗氧化物質,有非常多高水平的文章,這個文章很一般,國內許多實驗事都可以很容易實現。而且研究費用也比較少。

Food and Chemical Toxicology

Volume 47, Issue 8 , August 2009, Pages 2031-2036

Hepatoprotective effect of electrolyzed reduced waternext termagainst carbon tetrachloride-induced liver damage in mice

Chia-Fang Tsai 1, Yu-Wen Hsu 1, Wen-Kang Chen , Wen-Huei Chang , Cheng-Chieh Yen , Yung-Chyuan Ho and Fung-Jou Lu eCorresponding Author Contact InformationE-mail The Corresponding Author

Institute of Medicine, College of Medicine, Chung Shan Medical University, No. 110, Sec. 1, Jianguo N. Rd., Taichung 402, Taiwan

National Tainan Institute of Nursing, No. 78, Sec. 2, Minzu Rd., Tainan, Taiwan

School of Applied Chemistry, Chung Shan Medical University, No. 110, Sec. 1, Jianguo N. Rd., Taichung 402, Taiwan

School of Occupational Safety and Health, Chung Shan Medical University, No. 110, Sec. 1, Jianguo N. Rd., Taichung 402, Taiwan

Department of Nutrition, Chung Shan Medical University Hospital, No. 110, Sec. 1, Jianguo N. Rd., Taichung 402, Taiwan

Received 26 December 2008; 
accepted 18 May 2009. 
Available online 27 May 2009. 

 

Abstract

The study investigated the protective effect of previous termelectrolyzed reduced water next term(ERW) against carbon tetrachloride (CCl 4)-induced liver damage. Male ICR mice were randomly divided into control, CCl 4, CCl 4+ silymarin, and CCl 4+ ERW groups. CCl 4-induced liver lesions include leukocytes infiltration, hepatocyte necrosis, ballooning degeneration, mitosis, calcification, fibrosis and an increase of serum alanine aminotransferase (ALT), and aminotransferase (AST) activity. In addition, CCl 4also significantly decreased the activities of superoxide dismutase (SOD) and glutathione peroxidase (GSH-Px). By contrast, ERW or silymarin supplement significantly ameliorated the CCl 4-induced liver lesions, lowered the serum levels of hepatic enzyme markers (ALT and AST) and increased the activities of SOD, catalase, and GSH-Px in liver. Therefore, the results of this study show that ERW can be proposed to protect the liver against CCl 4-induced oxidative damage in mice, and the hepatoprotective effect might be correlated with its antioxidant and free radical scavenging effect.

 

作者台灣呂鋒洲教授介紹:

 http://medicine.csmu.edu.tw/front/bin/ptdetail.phtml?Part=914&Category=23

 

 

 

 


 

  

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