“氫水”還原健康 氫氣水可以治療牙周病(當心假氫水 破財又傷身)

氫氣水可以治療牙周病

已有 1545 次閱讀 2011-10-15 11:24 |個人分類:飲用氫氣水|系統分類:論文交流|關鍵字:xml office 牙周病 style black

 

氫氣水可以治療牙周病 www.h2-water.com

Hydrogen-rich water attenuates experimental periodontitis in a rat model.pdf

這是首次將氫氣用在口腔疾病的研究,這一研究的重要性是給我們提供了新的治療疾病類型,但這一疾病的重要性是發病率特別高,大部分人都會在某一階段存在一定的牙齒問題,而牙周病又占比較高的比例。這一疾病不僅適合進行臨床研究,而且適合個人嘗試。氫氣水非常容易獲得,對這個疾病,可以不需要飲用(但不限制),只需要漱口或含在口中就可以達到治療目的。從個人的體會看,口腔疾病,例如口腔潰瘍,直接漱口效果是非常理想的。希望很快將能看到人體試驗的報告。

 

本研究論文剛剛發表,目前尚沒有獲得全文,只是根據摘要的理解,可能存在偏差,引用請注意查看全文。

http://onlinelibrary.wiley.com/doi/10.1111/j.1600-051X.2011.01801.x/full

活性氧在牙周病的發展過程中發揮重要作用。因為氫氣分子可以作為活性氧清除劑,我們檢測了富氫氣水對大鼠牙周病的治療效果。將縫合線放在大鼠上頜骨臼齒周圍4周誘導牙周病,動物給飲普通水或富氫氣(對牙周病來講,反復用氫氣水漱口,或者用嘴含氫氣更好,不過動物可不會漱口)。結果發現:與正常無牙周病大鼠比較,飲用普通水的牙周病大鼠血清中活性氧隨著時間延長逐漸增加,同時齒槽骨出現中性顆粒細胞浸潤和骨質丟失(大概是牙周病的典型表現)。飲用富氫氣水牙周病大鼠血清中活性氧沒有增加, 4周後齒槽骨8oHdG和硝基酪氨酸表達減少(氧化損傷指標),而且中性顆粒細胞浸潤和骨質丟失也明顯受到保護。而且與炎症有關的信號傳導通路MAPK也受到明顯抑制。

結論:氫氣可以通過降低齒齦周圍炎症氧化損傷預防牙周病。

 

 


 

  

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Hydrogen-rich water attenuates experimental periodontitis in a rat model

Kenta Kasuyama, Takaaki Tomofuji, Daisuke Ekuni, Naofumi Tamaki, Tetsuji Azuma, Koichiro Irie, Yasumasa Endo Manabu Morita. Journal of Clinical Periodontology

 

Keywords:animal studies; antioxidants; hydrogen-rich water; oxidative stress; periodontitis

 

 

Abstract

Aim

 

Reactive oxygen species (ROS) contribute to the development of periodontitis. As molecular hydrogen can act as a scavenger of ROS, we examined the effects of treatment with hydrogen-rich water on a rat model of periodontitis.

Material & Methods

 

A ligature was placed around the maxillary molars for 4 weeks to induce periodontitis, and the animals were given drinking water with or without hydrogen-rich water.

Results

The rats with periodontitis which were treated with pure water showed a time-dependent increase in serum ROS level. Compared with the rats without periodontitis, the periodontitis-induced rats which were given pure water also showed polymorphonuclear leucocyte infiltration and alveolar bone loss at 4 weeks. Hydrogen-rich water intake inhibited an increase in serum ROS level and lowered expression of 8-hydroxydeoxyguanosine and nitrotyrosine in the periodontal tissue at 4 weeks. Such conditions prevented polymorphonuclear leucocyte infiltration and osteoclast differentiation following periodontitis progression. Furthermore, inflammatory signalling pathways, such as mitogen-activated protein kinases, were less activated in periodontal lesions from hydrogen-rich water-treated rats as compared with pure water-treated rats.

Conclusion

Consuming hydrogen-rich water might be beneficial in suppressing periodontitis progression by decreasing gingival oxidative stress.

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